I seem, for the time being, to be cancer free.

These are cancers or cancer cells.

And the cancer cell divides even more frequently and recklessly.

'Okay, like, yeah, I get why this is a cancer cell, and I get why this is a healthy cell.'

It is thought that a hypoxic, or oxygen poor environment, can increase cancerous cells.

Method One approach to cancer vaccination is to separate proteins from cancer cells and immunize cancer patients against those proteins, in the hope of stimulating an immune reaction that could kill the cancer cells.

The cancer cells, small, round and rapidly dividing, grew these arms and extensions.

That cancer cells pay a price for resistance is supported by several observations.

The cancer stem cells remain, and the tumor can return months or years later.

The cancer cells small, round and rapidly dividing, grew these arms and extensions.

Cancer cells pay a price when they evolve resistance to chemotherapy.

Up until now, we have been targeting cancers by targeting the cancer cells, trying to kill them.

But Wigler, besides looking at cancer cells, looked at normal cells, and made a really sort of surprising observation.

What we could do now is target those markers on the DNA, revert them to the original position, so that those cancer cells could act like normal cells.

This means that early cancerous cells should be sensitive to PARP inhibitors.

But neither radiation nor chemotherapeutic drugs target only cancer cells.

Let's look at a healthy liver cell, a healthy hair cell, and a cancerous cell.

The invasion of the lungs may lead to varying degrees of cell death.

This is because cancer stem cells are isolated from end stage tumors.

However, not every cell in the tumor has the ability to metastasize.

The second decade revealed the mechanism of the neuronal cell death.

Just as invasive species adapt to pesticides, most cancer cells adapt to therapies.

Our models show that in the absence of therapy, cancer cells that haven t evolved resistance will proliferate at the expense of the less fit resistant cells.

The patient receives an injection of a radio tracer that's taken up by rapidly proliferating tumor cells, but not by normal cells, and this is the key difference from mammography.

What fraction of tumor cells are CSCs and therefore need to be eliminated is not clear yet.

Cancer cells mutate, and they gain the ability to release lots of those angiogenic factors, natural fertilizer, that tip the balance in favor of blood vessels invading the cancer.

A number of studies have investigated the possibility of identifying specific markers that may distinguish CSCs from the bulk of the tumor (as well as from normal stem cells).

As this damage accumulates, it can lead to malfunctioning cells or cell death.

So we wanted to figure out how these ovarian cancer cells are becoming resistant to this drug called Cisplatin.

If it is possible to eliminate the cancer stem cell, then a potential cure may be achieved if there are no more CSCs to repopulate a cancer.

This protein plays an important role in angiogenesis and has been linked to cancer cell growth and Alzheimers progression.

Mammography relies on differences in the appearance of the tumor from the background tissue, and we've seen that those differences can be obscured in a dense breast.

Other types of white blood cells, such as lymphocytes, also undergo programmed cell death leading to an abnormally low concentration of lymphocytes in the blood.

Instead, it is used to provide an inhospitable environment for metastases so that they will not establish in other parts of the body.

The goal of radiation therapy is to damage as many cancer cells as possible, while limiting harm to nearby healthy tissue.

You put the cancer cells there, and they do go all over, they do this.

We're now learning to identify the cancer stem cells and identify those as targets and go for the long term cure.

That means that microenvironment and the context which surrounds those cells actually are telling the cancer gene and the cancer cell what to do.

Macrophages are the first cells infected with the virus, and this infection results in programmed cell death.

Researchers will need to establish the mechanisms by which cancer cells achieve resistance and what it costs them.

The first useful applications of nanomachines might be in medical technology, which could be used to identify and destroy cancer cells.

We've learned at Stanford and other places that we can discover cancer stem cells, the ones that seem to be really responsible for disease relapse.

Or where it gets its name from is if we were to pop this lysosome basically what happens is those digestive enzymes would go throughout the cell and would kill the cell, dissolve the cell.

The patient receives an injection of a radio tracer that's taken up by rapidly proliferating tumor cells, but not by normal cells, and this is the key difference from mammography.

There was another kind of cell that actually was sending out a toxin and contributing to the death of these motor neurons, and you simply couldn't see it until you had the human model.